Research Communications in

Substances of Abuse

 

 

 

Nutritional Status and Immune Function

in Cocaine and Heroin Abusers and

in Methadone Treated Subjects

 

N.D. Huggins (1), M.A. Khaled (1,2) P.E. Cornwell (2), and J.O. Alvarez (2,3)

 

Departments of Psychiarty and Behavioral Neurobiology,

(2) Nutrition Sciences, and (3) Public Health Sciences,

University of Alabama at Birmingham, University Station,

Birmingham, Al 35294, U.S.A.

 

Abstract

 

The plasma levels of some essential nutrients and the lymphocyte CD 4 to CD8 ratio were measured in four groups of

individuals that included: (a) cocaine and (b) heroin abusers, (c) methadone treated and (d) healthy subjects. Folate and

B-carotene levels were lower in the three drug groups while vitamin C was lower in the methadone and heroin subjects.

Vitamin E levels were boderline low in the methadone and cocaine groups. The methadone group also showed a

significantly higher level of lipid peroxidation which correlated well with the low values observed for the antioxidant nutrients.

Interestingly, the methadone group was the only one with a significantly reduced lymphocyte CD4/CD2 ratio.

 

 Introduction

 

Drug users, particularly intravenous drug abusers, are at signigicant risk of contracting HIV, hepatitis B and other infections.

Chandra, 1983 and numerous other authors have shown that malnutrition and infection are mutually aggravating through

their common association with immunity. Also, malnutrition has been linked to the reduction of lymphocyte T-helper ( CD4 )

to T-suppressor ( CD8 ) ratio ( Casey et al., 1983 ). On the other hand, addictive drugs of abuse have been shown not only to

produce significant nutritional aberrations ( Mohs, Watson and Leonard Green, 1990), but can also modulate

immunocompetence ( Donahoe and Falek, 1988).

 

Almost 25 years ago, methadone maintenance programs were started to prevent drug abusers from taking hard-core

narcotic drugs. Methadone, however, is a psychoactive drug, which ranks 17th on the Drug Abuse Warning Network, albeit

far below heroin. Immunological abormalities have been reported to occur frequently in heroin addicts and in methadone treated

subjects ( Kreek, 1990 ) although Novic et al., 1989 have reported that subjects on methadone programs for more than 11

years have normal immune functions in terms of CD4/CD8 T-cell ratio. Recently, however, Herbert et al., 1990 reported

nutritional abnormalities in terms of body mass index (BMI), hematocrit and hemoglobin concentration in patients on

a methadone maintenance program.

 

Nutritional status in general and specific nutrient imbalances in particular have been shown to affect the immune response. It is

plausible that nutritional deficits, common in drug abusers, may mediate or contribute to the deterioration of the immune

function in these individuals. Since there is a lack of information in the literature, a preliminary study was designed in order to

gain some insight into this problem.

 

Methods

 

Twenty-four human subjects participated in this study to compare four groups of six subjects: group 1, healthy subjects as

control; group 2, methadone treated for more than six months and less than five years; group 3, heroin addicts prior to entering

into the methadone maintenance program; and group 4, cocaine abusers. Noneof the subjects were HIV positive and the

human use protocol was approved by the Institutional Review Board of the University of Alabama at Birmingham (UAB).

Blood samples were withdrawn from each subject for the analysis of some essential micronutrients and to estimate their immune

status in terms of CD4/CD8 including lipid peroxidation ( as MDA ) and glutathione ( GSH ) levels.

 

Vitamin levels were determined using standard laboratory procedures in UAB's Nutrition Diagnostic Laboratory while the

measurements of CD4 and CD8 cells were made by the fluorescence tecnique ( Fahey, et al., 1984 ) using FITC conjugated

OKT4 and OKT8, respectively. The GSH level was measured spectophotometically, ( Beutler et al., 1963 ) and the lipid

peroxidation level in terms of malondialdehyde ( MDA ) production was estimated by HPLC ( Satoh, 1978 ). Statistical

calculations were performed by using ANOVA analysis and the significant differences are reported in the table.

 

 

Results and Discussion

 

The values in the Table show that the drug abusers in general have considerably lower folate and carotene levels. The vitamin C

status in both methadone and opiate groups were significantly lower than the control while the cocaine group showed a value

that falls in the lower side of the normal range. Although the vitamin E status was not statistically different, the values in the

methadone and cocaine groups were borderline normal.

 

In summary, the methadone group has more nutrient shortages than either the heroin or cocaine abusers, showing low values for

folate, and the antioxidant nutrients B-Carotine, vitamin C and vitamin E. Interestingly, the methadone group is the only one

that showed significantly elevated levels of MDA indicating an abnormally high level of lipid peroxidation. This supports the

notion that methadone treatment may be associated with an increased utilization of the body's antioxidant systems.

 

The finding that only the methadone-treated subjects have a lower lymphocyte CD4/CD8 ratio ( see table ) may suggest a

relationship, direct or indirect, between thir level of free radical production ( i.e., as evidenced by a high lipid perioxidation and

low levels of antioxidant nutrients ) and their immune function. It is not clear whether the methadone treatment is the cause of the

depletion of the antioxidant nutrients and immunodepression or whether the latter may result from the life-style and poor died of

these subjects.

 

Although the sample size in this preliminary study is small, the results presented here provide some significant information on the

nutritional status of drug abusers. Since intravenous drug abusers have a higher risk for contracting HIV infection and since

free radicals may be involve in the pathogenisis of viral infections, particularly AIDS ( Papadopulous-Eleopulos, 1988 ), the

potential interaction between nutritional status and immunodepression in these subject becomes even more important.

Cellular oxidative injury, mesured as an increased production of malondialdehyde ( Sonnerborg, et al., 1988 ) may play a role

in the progression of HIV infection. A double-blind, placebo-controlled study with sodium diethyldithiocarbonate, a potent

antioxidant, in patients with AIDS or AIDS-related complex have shown significant reduction in primary opportunistic

infections ( Lang, et al., 1988). Based on this information, a pertinent question may be asked: Will the nutritional and

immunological status of HIV-infected drug abusers who enter into methadone maintenance programs deteriorate fater, thus

leading to an earlier onset of full-blown AIDS disease? A larger, more thorough investigation of this relationship

is, therefore, warranted.

 

References

 

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61:882-888, 1963.

 

Casey J., Flynn W.R., Yao Jst., Fahey V., Pawlowski J. and Brogan J.J., Correlation of Immune and Nutritional Status with

Wound Complication of Immune and Nutritional Status with Wound Complications in Patients Undergoing Vascular

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Chandra, R.K. Nutrition, Immunity and Infection: Present Knowledge and Future Direction. Lancet, 1:688-691, 1983.

 

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Herbert Jr., Nichols S.E. and Kabat G.C. Indicators of Nutritional Status Among Clients from a New York City Methadone

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Kreek M.J. Immune Function in Heroin Addicts and Fromer Heroin Addicts in Treatment: Pre/Post AIDS epicemic.

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Novick D.M., Ochshorn M., Ghali V., Croxson T.S., Mercer W.D., Chiorazzi N. and Kreek M.J. Natural Killer Cell Activity and Lymphocyte Subsets in Parental Heroin Abusers and Long-term Methadone Maintenance Patients. J. Pharm. Exper. Ther., 250: 606-610, 1989

 

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Acta, 90: 37-43, 1978.

 

Sonnerborg A., Carlin G., Akerlund B. and Jarstrand C. Increased Production of Malondialdehyde in Patients With HIV

Infection. Scand. J. Infect. Dis. 20: 287-290, 1988.

 

 

 

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PJD Publications Ltd., P.O. Box 966, Westbury NY 11590, U.S.A.

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